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The study enrolled 132 patients with biopsy-proven nonalcoholic steatohepatitis (NASH), NASH cirrhosis, nonalcoholic fatty liver disease (NAFLD), and hepatitis B virus (HBV) cirrhosis. Fluctuation of ALT levels was not correlated with hepatic fibrosis. However, fluctuation of AST levels was positively correlated with the grade of hepatic fibrosis in patients with NASH. Moreover, fluctuation of AST levels was greater in the NASH patients with hepatic fibrosis than in the NASH patients without hepatic fibrosis. Furthermore, fluctuation of AST was greater in the NASH patients with HBV cirrhosis than in the NASH patients without cirrhosis. Fluctuation of AST was positively correlated with fluctuation of ALT levels in NASH patients, and ALT fluctuation was positively correlated with the degree of hepatic fibrosis in NASH patients. Our findings indicate that fluctuation of AST levels is an early marker for predicting the prognosis of NASH patients.The molecular mechanisms that regulate gene expression and cell fate are central to normal developmental processes and are disrupted in diseases such as cancer. In Drosophila, homeotic (HOM) genes control developmental fate by controlling the expression of selector genes. In the past decade, a large family of cell surface receptors that function in both immunity and development has been identified. The signaling mechanisms that mediate their activity are largely unknown. Our lab is interested in the mechanisms that regulate the activity of several immunoreceptor signaling proteins that function in the development I-DeClone 3.2.35.0 [32|64bit] 2-1. 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